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不知道為什麼又被封禁了，我這次已經詳細標註來源，甚至也翻譯了，結果還是被封禁，也同樣是被User:Mys_721tx，Mys_721tx管理員也都不願意對話，就直接封禁我了，上次我被封禁，我也附上相當可靠的資料來證實我沒有增加不實資料了，結果這次又封禁我增加不實資料，若認為我提供的資料有問題，請回應阿，都不正面回應，一直使用管理員的權限，而且我被封禁時間也沒有進行任何編輯，Mys_721tx就封禁我，也沒有詢問過我是否有進行編輯，這樣是正確的嗎? 若認為我添加不實資料，請回應我好嗎，也請先對話，不要我講了都不回應，然後直接用權限封禁我，我個人覺得，若管理員對於資料有不實，應該舉出相關文獻來反駁，而不是直接封禁使用者，我覺得維基百科如果要封禁使用者，必須管理員先舉出相關文獻反駁，在討論頁討論，你覺得資料哪裡不實要講啊，你也要提出相關文獻阿，上次他封禁我也反應出我提供的文獻討論，結果Mys_721tx也沒有回應，反而找其他理由封禁我，說我在封禁期間還進行編輯，可是我並沒有進行編輯阿，Mys_721tx就是一直想封禁我，我不知道為什麼他不想讓我編輯條目，也不知道為什麼他不願意正面討論，如果認為我增加的內容不合管理員的胃口，也不能這樣處理阿，若認為我增加不實資料拜託你先舉出相關文獻反駁阿，不是都不討論，就封禁，這樣到底怎麼編輯條目? 這樣怎麼進行條目的內容修改? 我要求Mys_721tx管理員提出可靠文獻反駁，Mys_721tx不討論不提供文獻資料想封禁就封禁，那到底要怎麼編輯這條目? 管理員也該提出相關文獻反駁阿WW（留言）

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千村狐兔（留言） 2020年4月12日 (日) 13:09 (UTC)回复
處理人︰請簽署

我沒有增加不實資料，上述申訴於1/12就申訴了，為什麼管理員沒有回覆? WW（留言） 2020年2月3日 (一) 02:25 (UTC)回复

吸毒比人類交配時更愉悅^{[來源請求]}，多巴胺增多意味著快感增多？--john doe 120@霍格華茲膜法學院^時間魔法 2020年2月3日 (一) 03:44 (UTC)回复

是的，多巴胺從中腦邊緣途徑進入NAcc殼的神經傳遞程度與愉悅感獎勵的激勵顯著性高度相關，The degree of dopamine neurotransmission into the NAcc shell from the mesolimbic pathway is highly correlated with the magnitude of incentive salience for rewarding stimuli。^[1]WW（留言）

^ Berridge KC (April 2012). "From prediction error to incentive salience: mesolimbic computation of reward motivation". Eur. J. Neurosci. 35 (7): 1124–1143. doi:10.1111/j.1460-9568.2012.07990.x. PMC 3325516. PMID 22487042. "Here I discuss how mesocorticolimbic mechanisms generate the motivation component of incentive salience. Incentive salience takes Pavlovian learning and memory as one input and as an equally important input takes neurobiological state factors (e.g. drug states, appetite states, satiety states) that can vary independently of learning. Neurobiological state changes can produce unlearned fluctuations or even reversals in the ability of a previously learned reward cue to trigger motivation. Such fluctuations in cue-triggered motivation can dramatically depart from all previously learned values about the associated reward outcome. ... Associative learning and prediction are important contributors to motivation for rewards. Learning gives incentive value to arbitrary cues such as a Pavlovian conditioned stimulus (CS) that is associated with a reward (unconditioned stimulus or UCS). Learned cues for reward are often potent triggers of desires. For example, learned cues can trigger normal appetites in everyone, and can sometimes trigger compulsive urges and relapse in addicts. Cue-triggered 'wanting’ for the UCS A brief CS encounter (or brief UCS encounter) often primes a pulse of elevated motivation to obtain and consume more reward UCS. This is a signature feature of incentive salience. Cue as attractive motivational magnets When a Pavlovian CS+ is attributed with incentive salience it not only triggers 'wanting’ for its UCS, but often the cue itself becomes highly attractive – even to an irrational degree. This cue attraction is another signature feature of incentive salience ... Two recognizable features of incentive salience are often visible that can be used in neuroscience experiments: (i) UCS-directed 'wanting’ – CS-triggered pulses of intensified 'wanting’ for the UCS reward; and (ii) CS-directed 'wanting’ – motivated attraction to the Pavlovian cue, which makes the arbitrary CS stimulus into a motivational magnet."

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[1] Berridge KC (April 2012). "From prediction error to incentive salience: mesolimbic computation of reward motivation". Eur. J. Neurosci. 35 (7): 1124–1143. doi:10.1111/j.1460-9568.2012.07990.x. PMC 3325516. PMID 22487042. "Here I discuss how mesocorticolimbic mechanisms generate the motivation component of incentive salience. Incentive salience takes Pavlovian learning and memory as one input and as an equally important input takes neurobiological state factors (e.g. drug states, appetite states, satiety states) that can vary independently of learning. Neurobiological state changes can produce unlearned fluctuations or even reversals in the ability of a previously learned reward cue to trigger motivation. Such fluctuations in cue-triggered motivation can dramatically depart from all previously learned values about the associated reward outcome. ... Associative learning and prediction are important contributors to motivation for rewards. Learning gives incentive value to arbitrary cues such as a Pavlovian conditioned stimulus (CS) that is associated with a reward (unconditioned stimulus or UCS). Learned cues for reward are often potent triggers of desires. For example, learned cues can trigger normal appetites in everyone, and can sometimes trigger compulsive urges and relapse in addicts. Cue-triggered 'wanting’ for the UCS A brief CS encounter (or brief UCS encounter) often primes a pulse of elevated motivation to obtain and consume more reward UCS. This is a signature feature of incentive salience. Cue as attractive motivational magnets When a Pavlovian CS+ is attributed with incentive salience it not only triggers 'wanting’ for its UCS, but often the cue itself becomes highly attractive – even to an irrational degree. This cue attraction is another signature feature of incentive salience ... Two recognizable features of incentive salience are often visible that can be used in neuroscience experiments: (i) UCS-directed 'wanting’ – CS-triggered pulses of intensified 'wanting’ for the UCS reward; and (ii) CS-directed 'wanting’ – motivated attraction to the Pavlovian cue, which makes the arbitrary CS stimulus into a motivational magnet."

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